Akciðer nodüllerinin yaklaþýk %95'i iyi huylu olmakla beraber, ABD'de onkolojik ölümlerin önde gelen en sýk nedeni akciðer kanseri olduðu için akciðer nodül-lerin türünü belirlemek gerekmektedir. Bir teþhis aracý seçerken, teþhis deðerinden ödün vermeden, yapýlan iþlem sayýsýný azaltabilmek önemlidir. Bu olgu sunu-munda, periferik yerleþimli bir akciðer nodülü ve mediastinal lenf nodlarýnýn biyopsisinde mevcut rad-yal ve lineer endobronþiyal ultrason rehberliðinde transbronþiyal iðne aspirasyonu ile beraber, robotik yardýmlý bronkoskopinin baþarýlý bir þekilde birlikte kullanýlmasý anlatýlmaktadýr. Tek bir iþlem ile hem periferik nodül hem de lenf nodülü, komplikasyonsuz olarak örneklendi. Periferik nodülde malingnite yoktu, ancak lenf nodülü Histoplazma ile uyumlu bulundu.

Some 95% of all detected lung nodules are benign, although it is essential to determine the underlying cause, given that lung cancer is the leading cause of oncological death in the United States. When choos-ing a diagnostic tool, reducing the number of proce-dures without compromising the diagnostic value is essential. This case report focuses on the successful use of the latest robotic-assisted bronchoscopy in combination with existing radial and linear endo-bronchial ultrasound-guided transbronchial needle aspiration for the biopsy of a peripherally located lung nodule and mediastinal lymph nodes. Combin-ing the two procedures allowed the peripheral nodule and lymph nodes to be sampled with no complica-tions in a single procedure. The nodule showed no cancerous growth, although the lymph nodes showed granulomas consistent with Histoplasma.

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Silikozisin ayýrýcý tanýsý sarkoidoz, berilyoz, hipersensi-tivite pnömonisi, malignite, tüberküloz ve diðer gra-nülomatöz enfeksiyonlarý içerir. Sarkoidoz etiyolojisi tam olarak bilinmeyen sistemik granülomatöz bir hastalýktýr ve silika, berilyum gibi mesleki maruziyet faktörlerin tetikleyici ajan olarak rol oynayabileceði düþünülmektedir. Eksik ve dikkatsiz bir meslek öyküsü birçok olguda pnömokonyoz yerine sarkoidoz tanýsý-na yol açabilir. Ek olarak bu iki hastalýðýn bir arada görülmesine veya bir diðerinin ötekinden önce sap-tamasý gibi durumlara da çeþitli makalelerde dikkat çekilmiþtir. Bu yazýda bir üniversite hastanesinin iþ ve meslek hastalýklarý bölümüne baþvuran beþ olgu üzerinden sarkoidoz ve silikoz birlikteliði tartýþýlmýþtýr. Tüm olgularda aktif akciðer tüberkülozu ve akciðer malignitesi dýþlandý. Tüm olgularýn biyopsi materyal-lerinin histopatolojik incelemeleri kazeifiye olmayan granülomatöz inflamasyon olarak rapor edildi. Bir olguda polarize mikroskopta ýþýðý çift kýran cisim gösterildi. Burada, bu olgular ýþýðýnda iyi alýnmýþ bir meslek öyküsünün sarkoidoz ve silikozisin ayýrýcý tanýsý ve etiyolojisini saptamadaki faydasýna dikkat çekmek amaçlanmýþtýr.

The differential diagnosis of silicosis includes sar-coidosis, berylliosis, hypersensitivity pneumonia, malignancy, tuberculosis and other granulomatous infections. Sarcoidosis is a systemic granulomatous disease with an unknown etiology, although it is thought that occupational exposure to such substanc-es as silica and beryllium may be a trigger. An in-complete occupational history may lead to a diagno-sis of sarcoidosis rather than pneumoconiosis in many cases, although various associations of these two diseases, such as the coexistence and detection of one before or after the other, have been reported in literature. The coexistence of sarcoidosis and sili-cosis is discussed in the present study with reference to five cases who applied to the Occupational Dis-eases department of a university hospital. Active pulmonary tuberculosis and lung malignancy were excluded in all cases, and histopathologic examina-tions of all samples were reported as non-caseating granulomatous inflammation. In the specimen of one case, a birefringent body was identified upon polar-ized microscopy. In the light of these cases, it is aimed to draw attention to the usefulness of a well-received occupational history in the differential diag-nosis and etiology of sarcoidosis and silicosis.

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Yapay hemoptizi oldukça nadirdir ve tanýsý doktorlarý zorlar. Kýrk yaþýndaki erkek mahkûmun dört yýldýr hemoptizisi vardý. Daha önce detaylý inceleme yapýlmýþ ancak hemoptizinin etiyolojisi belirlenememiþti. Son günlerde hemoptizi sýklýðý ve miktarý artmýþtý. Biz de radyolojik görüntüleme ve bronkoskopi dahil birçok tanýsal tetkikler yaptýk ancak kanamanýn lokalizasyonunu ve etiyolojisini belirleyemedik. Tam idyo-patik hemoptizi teþhisi koyacakken bazý þüpheli dav-ranýþlar ve çeliþkili ifadeler gördük. Psikiyatrik deðer-lendirmenin ardýndan hastaya ikinci eksen kiþilik bozukluðu ve yapay bozukluk tanýsý konuldu. Yapay hemoptizinin teþhisi zordur ve idiyopatik hemoptizi ile karýþtýrýlabilir. Detaylý bir muayene ile kanama yeri ve nedeni belirlenemeyen hemoptizi hastalarýnda düþünülmelidir.

Factitious hemoptysis is fairly rare condition, the diagnosis of which can be challenging to physicians. A 40-year-old incarcerated male with hemoptysis for four years had previously undergone a detailed inves-tigation, but the etiology of the hemoptysis could not be determined. The hemoptysis has increased in frequency and quantity, however, numerous diagnos-tic tests, including radiological imaging and bron-choscopy, has been unable to reveal the localization and etiology of the bleeding. As we were about to diagnose idiopathic hemoptysis, some suspicious behaviors and contradictory statements led us to conduct a psychiatric evaluation, and the patient was subsequently diagnosed with axis two personality disorder and factitious disorder. Factitious hemoptysis is difficult to diagnose and may be confused with idiopathic hemoptysis, and should be considered in patients with hemoptysis in which the location and cause of bleeding cannot be determined after a de-tailed examination.

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Bu olgu sunumunda, elektif artroskopik iþlem sonrasý nefes darlýðý geliþen, negatif basýnçlý pulmoner ödem (NBPÖ) tanýsý konan ve baþarýyla tedavi edilen bir hasta sunulmuþtur. Yirmi dokuz yaþýnda, herhangi bir týbbi öyküsü olmayan erkek hasta, askeri operasyon sýrasýnda geliþen rotator manþet yýrtýðý nedeniyle hastanemize baþvurdu. Genel anestezi altýnda artroskopik iþlem yapýldý. Ancak hastada ekstübasyondan 2 saat sonra dispne geliþti. Hastada satürasyon düþüklüðü ve takipne ile birlikte ve köpüklü pembe balgam görüldü. Bilgisayarlý tomografi pulmoner anjiyografide tromboemboli saptanmayýp, her iki akciðerde kardiyojenik olmayan pulmoner ödem düþündüren yaygýn buzlu cam opasiteleri saptandý. Hasta NBPÖ tanýsý konularak invazif olmayan mekanik ventilasyon, intravenöz furosemid ve inhale kýsa etkili betaagonistler ile tedavi edildi. Birkaç gün içinde belirgin bir iyileþme görülen hasta herhangi bir solunum semptomu olmadan taburcu edildi. Ekstübasyon sonrasý solunum yetmezliði geliþen kiþilerde hayatý tehdit edici bir tablo olan NBPÖ akla gelmelidir.

We report here on the successful management of a patient who developed dyspnea after an elective arthroscopic procedure and who was diagnosed with Negative Pressure Pulmonary Edema (NPPE). A 29-year-old male patient with no known medical history was admitted to our hospital due to a rotator cuff tear sustained during a military operation. An arthroscopic procedure under general anesthesia was performed; however, the patient developed dyspnea 2 hours after extubation. He was desaturated, tachypneic and began to produce frothy pink sputum. Pulmonary computed-tomography angiography revealed wide-spread ground-glass opacities in both lungs suggest-ing non-cardiogenic pulmonary edema without acute pulmonary thromboembolism. The patient was thus diagnosed with NPPE and treated with non-invasive mechanical ventilation, intravenous furosemide and inhaled short-acting beta-agonists. Significant recov-ery was observed in a couple of days, and the patient discharged after his respiratory symptoms abated. NPPE should immediately be suspected in individuals who develop respiratory failure following extubation, as it can be life threatening.

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Ýnhale anestezik sevoflurana maruz kaldýktan sonra yaygýn alveolar hemoraji (DAH) geliþen bir olguyu sunmayý amaçladýk. Burada sunulan hasta, jineko-masti nedeniyle plastik cerrahiye baþvuran 29 yaþýnda bir erkek idi. Genel anestezi altýnda intravenöz mida-zolam (4mg), fentanil ve inhale sevofluran ile kompli-kasyonsuz cerrahi uygulandý. Ameliyat sonunda ani hipoksemi ve masif kanama tespit edildi. Akciðer grafisinde bilateral yaygýn alveolar infiltratlar görüldü. Ameliyat sonrasý dönemde serum hemoglobin düzeyi 2,5 g/dl (13'ten 10,5 gr'a) düþtü. Hastaya 3 gün süreyle intravenöz olarak günlük metilprednizolon (1 gr) tedavisi verildi. Dördüncü günde hipoksemi dü-zeldi ve akciðer grafisinde alveolar infiltratlar kaybol-du. Literatürdeki sýnýrlý veriler, sevofluran ve DAH arasýnda bir iliþki olduðunu göstermektedir. Bizim olgumuzda DAH' geliþimine neden olan sebepler ve zemin hazýrlayan faktörlerin hiçbiri saptanmadý. Bu nedenle, DAH'dan inhale anestezik olarak sevoflu-ran'ýn sorumlu olduðu düþünülmektedir.

We present here a case of diffuse alveolar hemor-rhage (DAH) following exposure to inhaled anesthetic sevoflurane. A 29-year-old male was admitted to the plastic surgery department with gynecomastia. Sur-gery was performed without complication under gen-eral anesthesia with intravenously administered mid-azolam (4mg), fentanyl and inhaled sevoflurane. Immediate hypoxemia and massive hemorrhage was detected at the end of the operation, and a chest radiography revealed bilateral widespread alveolar infiltrates. The serum hemoglobin level dropped by 2.5 g/dl (from 13 to 10.5 gr) in the postoperative setting. The patient was treated with methylpredniso-lone (1 gr) administered intravenously daily for 3 days. The hypoxemia resolved and alveolar infiltrates on the chest radiograph disappeared on the 4th day. There is limited data in literature reporting on the association between sevoflurane and DAH. None of the predisposing factors or causative reasons for DAH were detected in our case, and so it can be conclud-ed that the use of sevoflurane as an inhaled anesthet-ic was responsible for the DAH.

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Dalak tüberkülozu, literatürde zaman zaman, çoðun-lukla çeþitli risk faktörleri olan baðýþýklýðý baskýlanmýþ bireylerde tanýmlanmýþtýr. Dalak tutulumu ve spleno-megalinin olmasý, bu tür Mycobacterium tuberculosis enfeksiyonunun teþhis edilmesini zorlaþtýrýr. Spleno-megali ile birlikte mediastinal ve abdominal lenfade-nopatileri olan ve lenfoma ön tanýsý ile araþtýrýlan, önemli bir týbbi öyküsü olmayan 56 yaþýnda kadýn hasta ateþ, halsizlik yakýnmasý ile baþvurdu. Alýnan plevral ve dalak biyopsisinde granülomatöz inflamas-yonun ortaya çýktý. Burada, dalak tüberkülozunun eþlik ettiði tüberküloz lenfadenit ve plörit olgusunu sunuyoruz Tüberküloz, nedeni bilinmeyen ateþ ve lenfadenopatilerin eþlik ettiði hastalýklarda ayýrýcý taný olarak hala akýlda tutulmalýdýr.

Spleen tuberculosis has occasionally been described in literature, mostly in immunocompromised patients with various risk factors. The presence of spleen in-volvement and splenomegaly makes this type of My-cobacterium tuberculosis infection difficult to diag-nose. A 56-year-old woman who had mediastinal and abdominal lymphadenopathies with splenomeg-aly and was investigated with a pre-diagnosis of lymphoma, and with no significant medical history, presented with complaints of fever and malaise. Here, we present a case of tuberculous lymphadenitis and pleuritis accompanied by splenic tuberculosis with the appearance of granulomatous inflammation on pleu-ral and spleen biopsy. Tuberculosis should be kept in mind as a differential diagnosis in diseases accom-panied by a fever of unknown origin and lymphade-nopathy.

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Down sendromunda solunum yolu enfeksiyonlarý sýk olarak görülmektedir. Diabetes mellitusu da olan Down sendromlu 24 yaþýndaki erkek hasta, ateþ ve kusma þikayetleri ile gittiði dýþ merkezde akciðer ap-sesi tanýsýyla antibiyotik tedavisi almýþ, ancak semp-tomlarýnýn gerilememesi üzerine tarafýmýza sevk edil-miþtir. Radyolojik incelemede akciðer apsesinde dü-zelme olmasýna raðmen ateþ, kusmanýn devam etme-si ve baþ aðrýsýnýn da baþlamasý üzerine yapýlan ince-lemelerde hastanýn beyin apsesinin de olduðu sap-tandý. Özellikle altta yatan ek hastalýðý olan akciðer apseli olgularda beyin apsesi gibi ek apse odaklarý açýsýndan dikkatli olunmalýdýr.

Respiratory tract infections are common in Down syndrome. A 24-year-old male patient with Down syndrome with concurrent diabetes mellitus under-went antibiotic treatment with a diagnosis of lung abscess in an external center with complaints of fever and vomiting, but was referred to us after his symp-toms did not regress. Despite the improvement in the lung abscess noted in a radiological examination, the patient was identified with a brain abscess upon an examination due to the continuation of fever, vomit-ing and the onset of headache. Particular attention should be paid to additional abscess foci such as brain abscess in cases with lung abscess with an underlying comorbidity.

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Primer plevral sinovyal sarkom tanýlý 29 yaþýnda erkek hastanýn, geliþinde herhangi bir þikayeti yoktu, sigara içmiyordu ve özgeçmiþinde özellik yoktu. PA akciðer grafisinde, göðüs duvarýna bitiþik düzgün sýnýrlý dev kitle izlendi. Toraks BT’sinde; sað hemitoraks latera-linde, diffüz amorf kalsifikasyon içeren solid kitlesel lezyon tespit edildi. PET/BT’de, tanýmlanan lezyonda SUVmax 5.0 olan F18-FDG tutulumu saptandý. Tru-cut biyopsi sonucunda, histolojik olarak monofazik, yoðun hücresel ve birbiri üzerine geçen fasiküllerin oluþturduðu iðsi hücreler, SYT-SSX1 füzyon (X;18)(p11.23;q11) saptanmasý üzerine sinovyal sarkom tanýsý kondu. Cerrahi ve kemoradyoterapi sonrasýnda 1 yýldýr stabil olarak onkoloji merkezinde takip edilmektedir. Bu yazý, nadir görülen ve agresif bir tumor olan primer plevral sinovyal sarkomun taný basamaklarý ve iyi sonuçlar alýnabilmesi için hýzlýca tedavisinin baþlanmasýna dikkat çekmeyi amaçlamak-tadýr.

We present here the case of a 29-year-old male with primary pleural synovial sarcoma. The patient had no complaints, no significant medical history and did not smoke. A well-circumscribed giant mass, adjacent to the chest wall was observed on a PA chest X-ray, with a solid mass lesion in the lateral right hemithorax with diffuse amorphous calcifications noted on thorax computed tomography. An F18-FDG uptake with a SUVmax of 5.0 was detected in the mass defined by PET/CT. A Tru-cut biopsy revealed monophasic as histological subtypes composed of a dense cellular and interlaced fascicular proliferation of spindle cells, and SYT-SSX1 fusion (X;18)(p11.23;q11) consistent with synovial sarcoma. The patient was followed-up in the oncology center after surgery and chemoradio-therapy, and has been stable for 1 year. This paper draws attention to a diagnostic approach to primary pleural synovial sarcoma, which is a rare and aggres-sive tumor, and the need for the prompt initiation of treatment for an improved outcome.

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Kommerell divertikülü (KD), genellikle aberran bir sað subklavyen arterin (ARSA) veya aberran sol subklav-yen arterin (ALSA) çýkýþýnda bulunan aortik arkýn distal kýsmýnýn konjenital dilatasyonudur. Bu olguda, sað taraflý aortik ark (RAA) ile beraber ligamentum arteri-yozumu (LA) olan ve KD'den kaynaklanan ALSA'sý olan 40 yaþýnda bir erkek hastayý tanýmladýk. Polikli-niðimize baþvurmuþ bu hastanýn son bir yýl içinde devam eden nefes darlýðý ve yutma güçlüðü öyküsü vardý. Kliniðimizde hastaya, ligamentum arteriyozum-lu RAA'da ALSA düzeyinde KD’ü göstermek için akci-ðer tomografisi çekildi. Semptomlar nedeniyle KD ve LA’nýn cerrahi rezeksiyonu planlandý. Ýnen aortadan KD rezeke edildi ve ALSA sol karotid artere transfer edildi. Hasta ameliyattan 6 gün sonra komplikasyon-suz olarak taburcu edildi. Ameliyattan sonra hastanýn dispne ve disfajisi tamamen kayboldu.

Kommerell's diverticulum (KD) is a congenital dilata-tion of the distal portion of the aortic arch that is usually located at the origin of an aberrant right subclavian artery (ARSA) or the aberrant left subclavi-an artery (ALSA). We describe here a 40-year-old male patient with a right-sided aortic arch (RAA) with ligamentum arteriosum (LA) and ALSA originating from the KD. The applied to our outpatient clinic with a history of dyspnea and dysphagia that had persist-ed for the last year. The patient underwent chest computed tomography (CT) in our clinic, revealing ALSA-level KD in RAA with ligamentum arteriosum. Surgical resection of KD and LA was planned based on the symptoms. NE was resected from the descend-ing aorta and ALSA was transferred to the left carotid artery, and the patient was discharged 6 days after the operation without complications. After the opera-tion, the patient's dyspnea and dysphagia subsided completely.

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Aberran sað subklavyen arter (ASSA) konjenital bir anomali olup, sýklýkla asemptomatik seyreden ve nadir görülen bir anomalidir. Eriþkin ASSA’lý hasta-larda en sýk klinik baþvuru semptomu disfaji olup nadirde olsa solunum yakýnmalarý ile de baþvurabilir-ler. Yutma güçlüðü ile baþvuran 79 yaþýndaki bir hasta çekilen toraks ve boyun BT, özofagografi ile taný konularak sunulmuþtur. Hasta yutkunamama ve öksürük þikayeti ile baþvurduðu merkezlerde pnömoni tedavisi görmüþ, çekilen grafiler sonrasý mediastinal kitle þüphesiyle sevk edilen hastaya kontrastlý bilgisa-yarlý tomografi ile ASSA tanýsý konulmuþtur. Sonuç olarak yutma güçlüðü ile baþvuran hastalarda ASSA buna neden olan nadir bir sebepte olsa ayýrýcý tanýda yer almalýdýr.

Aberrant right subclavian artery (ARSA) is a rarely seen congenital and often asymptomatic anomaly. The most common clinical presenting symptom in adult ARSA patients is dysphagia, while patients may rarely present with respiratory complaints. We present here the case of a 79-year-old patient with dysphagia who was diagnosed based on thorax and neck CT and esophagography findings. The patient was treat-ed for pneumonia in the centers to which she applied with complaints of cough and inability to swallow, and was subsequently referred to us with the suspi-cion of a mediastinal mass after radiograms were obtained. The diagnosis of ARSA was made based on contrast-enhanced computed tomography. With this in mind, ARSA should be included in the differential diagnosis of patients presenting with dysphagia, despite it being a rarely seen etiology.

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HIV (Ýnsan Ýmmün Yetmezlik Virüsü) immün sistemi zayýflatan, AIDS (Kazanýlmýþ Baðýþýklýk Yetersizliði Sendromu) hastalýðýna yol açan viral bir enfeksiyon-dur. Ýmmün sistemin zayýflamasý ile birlikte, tüberkü-loz, PCP (Pneumocystis jiroveci pnömonisi), CMV (sitomegalovirüs), kandida gibi fýrsatçý enfeksiyonlara olanak saðlamaktadýr. En yüksek mortaliteyi oluþturan durumlar fýrsatçý enfeksiyonlardýr. Bu durum AIDS hastalarýnda fýrsatçý enfeksiyonlara yönelik tarama ve profilaksi uygulanmasýný gerekli kýlmýþtýr. Tüberküloz ile HIV birlikteliði uzun zamandýr bilinmekte ve prog-nozu kötüleþtirmektedirler. PCP ise günümüzde man-tar olarak kabul edilen P. jirovecii etkenli fýrsatçý bir pnömonidir. Covid -19 pandemisi ile birlikte de AIDS hastalarýnda yeni bir mortalite sebebi ortaya çýkmýþtýr. AIDS hastalarýnýn bu fýrsatçý enfeksiyonlara karþý taranmasý, profilaktik tedavi almasý ve erkenden tedavi almalarý hayati önem taþýmaktadýr. Ancak HIV pozitifliði rastlantýsal olarak saptanan kiþilerde tarama ve profilaksi gecikmesinden ötürü mortalite yüksek seyredebilmektedir. Bu yazýda Covid-19 pnömonisi þüphesi ile hastanemize baþvuran bir hastanýn HIV pozitif tespit edilip PCP ve takibinde akciðer tüberkü-lozu saptanmasý anlatýlmaktadýr.

HIV (Human Immunodeficiency Virus) is the virus that causes AIDS (Acquired Immune Deficiency Syndrome), while PCP (Pneumocystis jiroveci pneumonia), tuber-culosis, CMV (Cytomegalovirus) and candidiasis are the OIs (opportunistic infections) occurring due to immune deficiency. OIs, and bacterial pneumonias in particular, are the most common causes of mortality, which makes the screening and prophylactic therapy for OIs necessary. The synergy between tuberculosis and HIV has long been known, and worsens the prognosis. PCP is an OI that is caused by a fungus named P. jiroveci. COVID-19 has emerged as a new cause of death among AIDS patients. Screening and prophylactic therapy for Ols is vital for patients with AIDS, however, mortality may be high due to delays in screening and prophylaxis in those whose HIV positivity is detected coincidentally. We report here on a patient who applied to our hospital with suspected COVID-19 pneumonia who was found during follow-up to be HIV positive with PCP and pulmonary tuber-culosis.

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COVID-19, asemptomatikten þiddetli semptomlarla giden hastalýða kadar geniþ bir klinik spektrum göste-ren, çoðunlukla solunum yollarýný etkileyen yeni tip koronavirüs enfeksiyonudur. Hastalýkta temel etkile-nen bölge solunum sistemi olsa da, pandeminin erken evrelerinden itibaren bazý hastalarda, baþ aðrý-sý, baþ dönmesi ve kas aðrýlarý gibi nörolojik semp-tomlar da bildirilmiþtir. COVID-19 semptomlarý ve komplikasyonlarý, hem periferik hem de santral sinir sistemini ve iskelet kaslarýný etkileyebilir. Epileptik nöbet, COVID-19'un nadir bir belirtisidir. Biz de COVID-19 ensefalopatisi nedeniyle, hastaneye epi-leptik nöbet ile baþvuran bir kadýn hastayý sunuyoruz.

COVID-19 is a new type of coronavirus infection with a wide clinical spectrum, ranging from asymptomatic to severely symptomatic, and that mostly affects the respiratory tract. Although the respiratory tract is the primary area affected by the disease, neurological symptoms such as headache, dizziness and muscle ache have also been reported in some patients since the early stages of the pandemic. COVID-19 symp-toms and complications can affect the peripheral and central nervous systems as well as the skeletal mus-cles, while epileptic seizure is a rare manifestation of COVID-19. We present here the case of a female patient admitted to hospital with epileptic seizure due to COVID-19 encephalopathy.

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Post- COVID pulmoner fibrozis ve tromboemboli geliþimine dair çok sayýda yayýn olmasýna raðmen, bronþektazi geliþimi ile ilgili literatürde sýnýrlý sayýda yayýn ve olgu sunumlarý bulunmaktadýr. Biz de bu çalýþmamýzda post-COVID 11. ayda bronþektazi sekeli saptanan olgumuzu sunmayý amaçladýk. Kýrk dokuz yaþýnda, erkek, diabetes mellitus ve hipertansi-yon tanýlý, non-smoker hasta, efor dispnesi ile baþ-vurdu. On bir ay önce aðýr COVID-19 pnömonisi ve solunum yetmezliði ile hastanede 1,5 ay süreyle yatý-rýlarak izlenmiþti. Favipiravir, pulse metilprednizolon ve geniþ spekturumlu antibiyotik verilen hastanýn invazif mekanik ventilatör ihtiyacý olmamýþtý ve sekon-der bakteriyel enfeksiyon saptanmamýþtý. Toraks BT‘sinde eski BT’leri ile kýyaslandýðýnda fibrotik deði-þiklikler kaybolmasýna raðmen bronþektazisinin sebat ettiði görüldü. Gelecek yýllarda bronþektazi etyoloji-sinde ilk sorgulayacaðýmýz nedenlerden biri COVID-19 geçirme öyküsü olabilir.

Although there have been many studies determining the occurrence of post-COVID pulmonary fibrosis and thromboembolism, there are a limited number of studies and case reports in literature on the develop-ment of bronchiectasis. The present study presents a case of bronchiectasis sequel in the post-COVID 11th month. A 49-year-old male, non-smoker with diabe-tes mellitus and hypertension was admitted with exer-tional dyspnea. The patient had been followed up in the hospital 11 months earlier for 1.5 months with severe COVID-19 pneumonia and respiratory failure for which he was treated with Favipiravir, pulse methylprednisolone and broad-spectrum antibiotics. There was no need for invasive mechanical ventila-tion, and no secondary bacterial infection was de-tected. Compared to the previous CT, a chest CT revealed that bronchiectasis had persisted despite the disappearance of fibrotic changes. In the coming years, one of the first questions raised regarding the etiology of bronchiectasis may be the patient’s COVID-19 history.

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